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In many mammals the optic tract terminates on the superficial layers of the superior colliculus (SC) which projects axons to the paraventricular thalamic nucleus (PVT). In the rat these axons consist of a thick (approximately 1.0 mm) axonal collaterals that extend in the SC, and a thinner (approximately 0.1 mm) dendritic collaterals that course through the deep layers of the SC. In the cat, the bidirectional connection between SC and PVT is terminated in the deep layers of the SC. We thus aimed to determine if this terminal retraction is associated with a loss of function in the SC-PVT pathway in order to assess the behavioural consequences of the lesion. Extracellular recording and microiontophoretic injections of retrograde tracers were made in the SC of adult cats anaesthetized with chloralose/N2O/O2. The lesion was produced by mechanical transection of the optic tract in adult cats. The re-appearance of the bidirectional connection between the SC and PVT was confirmed in 8 cases by the re-entry of fluorescent tracer to the PVT, 6 weeks after the lesion. There were no changes in the number of cells activated by electrical stimulation of the SC or PVT, and no change in the excitatory or inhibitory components of the responses in the PVT. We conclude that there is no loss of function of the SC-PVT pathway in the cat lesioned in adulthood, although we cannot rule out a loss of effect of the SC-PVT connection in the developing animal.The U.S. Senate has approved an amendment adding a provision to a $1 trillion infrastructure bill aimed at providing $50 billion in federal assistance for states that create a permit-to-possess registry for commercial marijuana.

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